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CANCER MENUbar CANCER MENUbar GSE 1-2-3 Cancer Cure Plan - Enzymes break down fibrin (cancer's protective "cloaking device")

(1) Cancer NOT Allowed

Systemic proteolytic enzymes ”Decloak” cancer cells

To find out which foods have the highest enzymes and ways to boost systemic proteolytic enzymes and also their efficiency in the body:

How to boost body's systemic proteolytic enzymes

Tumor cells have a protective  mucus/fibrin coating acting as a “Cloaking” device

Tumor cells have a proteinous mucus/fibrin coat designed to protect them from the body's immune system

–   The mucus/fibrin coat acts as a “Cloaking device” - preventing cancer cells from being recognized by the immune system's white blood cells (WBCs: macrophages, neutrophils), and thus making them immune to attack by natural killer (NK) cells. Mucus is a glycoprotein (sugar+protein), fibrin is a a protein in the blood, which enables blood to clot.

–   The sticky fibrin coat is ~15 times thicker than the fibrin which surrounds healthy cells – and is revealed by histochemical and microscopic examination.

Egyud LG, Lipinski B. Significance of fibrin formation and dissolution in the pathogenesis and treatment of cancer. Med Hypotheses. 1991 Dec;36(4):336-40.

Massimo Cardinali et al. Interaction of Fibrinogen with Murine Melanoma Cells: Covalent Association with Cell Membranes and Protection against Recognition by Lymphokine-activated Killer Cells. Cancer Res December 15, 1990 50; 8010 Study Link

Systemic proteolytic enzymes break down the fibrin “shield” - effectively “De-cloaking” the cancer cell!

–   WBC's use mainly elastase and cathepsin G enzymes in fibrinolysis

Plow EF. The major fibrinolytic proteases of human leukocytes. 1980 Jun 5 Biochim Biophys Acta.;630(1):47-56. PubMed


Systemic proteolytic enzymes boost immune function

John Beard believed pancreatic enzyme deficiency impairing immune function was the root cause of cancer


Enzymes boost immune system by increasing cancer cell-killing cytokines - particularly interferon and tumor necrosis factor-α (TNF-α). TNF-αmade by macrophages triggers death of cancer cells by locking on to and activatinga receptor site (called the “Death Domain”) on a cancer cell, which:

✔ Forces the cancer cell to send out chemicals that help the immune system to locate it

✔ Triggers apoptosis (“cellular suicide”)

✔ Causes the death domain to send out signals to I.S. neutrophils ,which travel through mucus, lymph or the linings of healthy organs to join the attack

✔ Inhibits tumorigenesis and viral replication

✔ Induces cachexia

–   Systemic proteolytic enzymes remove the cancer cell debris to give immune system cells a clear line-of-fire - Eventually the macrophage (“Big Eater”) engulfs and “eats” the cancer cell. A cluster of just a few thousand cancer cells are easily dealt with by the above process. However, if the numbers become overwhelming, there are so many ”dying” cancerous cells that the I.S. can't find the tumor. This is when the I.S. needs some help from enzymes to clear away the debris so it can focus on the “not dead” cancer cells. Proteolytic enzmes break down protein. A 1920's physician Dr. Max Wolfe found that cancer patients tend to lack proteolytic enzymes in their GI tract, not necessarily because they were not consuming or producing enough enzymes, but also because the enzymes had “gone off to war” fighting cancer cells . German studies demonstrate a 12-fold increase in the potency of macrophages and killer cells by increasing systemic enzymes.

Systemic proteolytic enzymes help prevent metasases

Systemic proteolytic enzymes help prevent metasases by these mechanisms:

✔  By boosting immune function to eliminate cancer cells

✔  Also, by hindering their ability to adhere to endothelial cells (vessel wall lining)

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