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Cholesterol does NOT cause CHD

Cholesterol and Saturated Fat do NOT cause CHD

The Scam of the Century!

You do not need Lipitor™, Zocor™, Pravachol™, Mevacor™, Crestor™ or any other cholesterol-lowering statin drug, because the root CAUSE of coronary heart disease and other ischaemic cardiovascular diseases is NOT cholesterol - in  order to sell you a solution to this frightening “death threat”, you have been sold the multi-billion dollar scam of the century.

BTW, you also don't need beta blockers(reduce heart rate by diminishing the effects of adrenaline and other stress hormones) or calcium channel blockers (which lower blood pressure by relaxing the muscles in artery walls by blocking the entrance of calcium into muscle cells, but also block essential heart and blood vessel functions).

Cholesterol and saturated fats are NOT the “Darth Vader” of Cardiovascular Disease

Elevated blood cholesterol and saturated fats have been showcased as the major culprits causing arterial plaque, with its consequential strokes and heart attacks,

but read on . . .

The Diet-Heart Hypothesis is wrong

“The diet-heart hypothesis has been repeatedly shown to be wrong, and yet, for complicated reasons of pride, profit and prejudice, the hypothesis continues to be exploited by scientists, fund-raising enterprises, food companies and even governmental agencies. THE PUBLIC IS BEING DECEIVED BY THE GREATEST HEALTH SCAM OF THE CENTURY.”

–   George Mann, SsD, MD, Former Co-Director, Framingham Heart Study

Still unproven - the diet–heart hypothesis relies on two parts:

PART (1)

Consuming dietary cholesterol/saturated fats increases levels of cholesterol in the blood


►  ►  ► 

This is simply not true!


This misguided idea is based on the "lipid hypothesis", developed in the 1950s by Ancel Keys - In an attempt to prove a link between dietary fats and CHD, Keys cherry-picked data for his published analysis from only 6 countries, out of the 22 countries for which data was available at the time. This extremely flawed analysis began the push for the “margarine era”and the “low fat”diet by government health agencies, resulting in the obesity and diabetic epidemic of today, not-to-mention making tidy profits for the polyunsaturated vegetable seed oil industry.



Short Youtube clip from the documentary “Fat head” shows how the flawed lipid hypothesis began

And yet, people are still encouraged to adjust their diet to include less saturated fat and cholesterol (a task that pharmaceutical companies know that we humans are doomed to fail and will thus turn to their drugs to solve the problem),, and yet . . ..

–   Oxidized cholesterol and saturated fat typically constitute less than a quarter of arterial plaque, and cholesterol levels are actually not much affected by diet - Most people can give up steak, eggs, cheese, and butter, and their cholesterol may not budge a single point. This is because the body can make as much as 1500 grams of cholesterol a day, which may be more than 6 times the amount in the diet.

–   The body has numerous essential uses for cholesterol - and when you consume it, the body doesn't need to make as much.

–    Even Ancel Keys, the originator of the Heart-Diet Hypothesis (which was based on studies feeding oxidized cholesterol to rabbits, causing their cholesterol levels to skyrocket), admits there is no connection between dietary cholesterol and blood cholesterol:

“There's no connection whatsoever between the cholesterol in food and cholesterol in the blood. And we've known that all along. Cholesterol in the diet doesn't matter at all unless you happen to be a chicken or a rabbit.”

–    Ancel Keys

Rabbits are herbivores . . . they are not supposed to eat cholesterol, but in contrast, we've been eating it for thousands of years.

Analyses of 60 years of studies pronounced a verdict of “Not Guilty” in the case implicating “Saturated fat” with Heart Disease

–   23 nutritional factors were found to be moderately/strongly associated with heart disease, but . . . Saturated fat was not one of them! -found researchers from McMaster University in 2009, who undertook “a systematic review of evidence linking a wide variety of nutritional factors and heart disease”, publishing their results in the Archives of Internal Medicine. They analyzed146 researchstudies involving millions of people over a span> 60 years.

–   The combined results of several epidemiologic studies showed that there is no significant evidence for concluding that dietary saturated fat is associated with an increased risk of CHD or CVD - concluded a study published in the Jan. 2010 Journal of Clinical Nutrition, which analyzed the association of dietary saturated fat with risk of coronary heart disease, stroke and cardiovascular disease in 21 epidemiological studies (look for associations between things). 5–23 years of follow-up of 347,747 subjects determined that 11,006 developed CHD or stroke. Consideration of age, sex, and study quality did not change the results.


–    A Medical Research Council survey showed that men eating butter ran half the risk of developing heart disease as those using margarine

Nutr Week, Mar 22, 1991, 21:12:2-3

–   As heart-disease rates skyrocketed in the mid-1900s, animal fat consumption was actually going down – while vegetable oil (with its inherent excessive omega-6 content) was being consumed in dramatically increasing amounts.

–   Half of all heart-attack victims have normal or low cholesterol - autopsies performed on heart-attack victims routinely reveal plaque-filled arteries in people whose cholesterol level was low.

The medical profession and articles all over the internet continue to espouse the largely unfounded dietary guidelines such as those on the Heart and Stroke Foundation website:


"Limit foods high in saturated fat, trans fat and/or cholesterol, such as whole-milk dairy products, fatty meats, tropical oils, partially hydrogenated vegetable oils and egg yolks. Instead choose foods low in saturated fat, trans fat and cholesterol. Here are somehelpful tips:

–    Include: fat-free and low-fat milk products, legumes (beans), skinless poultry and lean meats.

–   Choose fats and oils: such as liquid and tub margarines, canola, corn, safflower, soy bean and olive oils.”


Except: You should minimize intake of trans fats / partially hydrogenated vegetable oils, found in high-heat-processed polyunsaturated vegetable oils (most all grocery-store offerings E.g. Canola, soybean, corn, safflower oils) and processed foods containing these oils (including margarine), including restaurant foods fried in them

(Must be they don't subscribe to Archives of Internal Medicineor American Journal of Clinical Nutrition).

After 60 years of trying, the proponents of the diet-heart hypothesis have still not been able to prove that dietary saturated fat or cholesterol has anything to do with heart disease. Attempting to lower your risk of getting heart disease by reducing intake of saturated fat is as helpful as throwing a bucket of water at a forest fire.

For supporting studies, see:

Dietary fat / Cholesterol has little effect on blood cholesterol levels

To appreciate the many vital functions of cholesterol, see:

Cholesterol - Our Hero!

PART (2)

Having elevated cholesterol levels in the blood causes heart disease

This should read: “is weakly associated with” instead of “causes”- which has quite a different meaning.

There have been literally hundreds of studies done on the association between blood cholesterol and heart disease. Some found a weak association, others a weak disassociation.

Large Framington Heart Study debunked the “Blood cholesterol causes heart disease”myth – a Massachusetts, U.S. study ongoing since 1948, directed by National Heart Institute; originally enrolled 5209 men and women, who had no signs of CVD; participants tested every 2 years. In 1971, 5124 of the original participants' adult children and their spouses were also enrolled in the study.

This most famed study and the onemost quoted to support the hypothesis simply concluded that having high blood cholesterol levels was the best indicatorfor heart disease risk. The correlation factor between blood cholesterol and heart disease was 0.36, an insignificant amount in a study this size, (BTW - no other study has found a higher correlation). Considering the results, what they should have concluded, is that none of the factors they looked at predicted heart disease and blood cholesterol was not a significant predictor of heart disease. They did not conclude that cholesterol was the cause of heart disease, which is what has been erroneously quoted. (Maybe, someone needed to justify the mega-cost of the study?)

The study actually found that over a third of coronary heart disease occurs in people who do NOT have particularly high total cholesterol levels

“An analysis of cholesterol values . . . in 1,700 patients with atherosclerotic disease revealed no definite correlation between serum cholesterol levels and the nature and extent of atherosclerotic disease.”

- Michael DeBakey, MD, Famous heart surgeon

Grossly unscientific logic is confusing a weak correlation with cause - elevated blood cholesterol being weakly correlated with heart attacks does not mean cholesterol causes heart attacks. By way of illustration - gray hair is correlated with getting older, but it doesn't mean gray hair is the cause of getting old.

With the obvious flaws in the theory, some amendments were necessary - Enter center stage - HDL and LDL cholesterol.LDL (the supposed “bad”guy) carries cholesterol totissues, and if it hangs around it ends up getting oxidized. HDL (called the “good”guy), carries excess cholesterol back to the liver fromthe tissues. The amended theory is that the oxidation of LDL in the blood begins the atherosclerosis process, leading to heart disease - it makes sense. Keep your LDL low and your HDL high and you're good to go! The theory vindicates most cholesterol, but vilifies LDL cholesterol (and its buddy saturated fat).

HDL, LDL and total cholesterol values do provide some secondary indicators of a problem

Total blood cholesterol is calculated as:

HDL value + LDL value + Trigycerides/5

The total cholesterol value on its own doesn't tell you a whole lot, unless its >300, which does indicate a high risk of CHD. (People with 250 can be at low risk for heart disease due to their HDL levels. Conversely,people with a cholesterol level < 200 can be at a very high risk of heart disease).

Two ratios do provide good indicators of a problem:

HDL value/ Total cholesterol - percentage should be >24%;


Triglyceride / HDL - percentage should be <2%.

However, lowering cholesterol is NOT ENOUGH to prevent death from cardiovascular disease - The chart here shows the results of eight large worldwide trials comparing control groups to those of treatment groups lowering their cholesterol.

The people lowering their total cholesterol (particularly LDL cholesterol) levels, showed only an average 25-35% reduction in number of cardiovascular events, compared to the control groups.

(a)  A large number of patients who lower their LDL cholesterol with a holesterol-lowering therapy continue to have cardiovascular events,


(b)  80% of people who develop CVD have the same blood cholesterol values as those who do not develop CVD or incur a heart attack.

Neither part (1) or part (2) of the diet-heart hypothesis could be proven - This left just a weak association with high LDL blood levels, but the parties who stood to gain from the theory simply jumped from this weak association - to a cause. The general public were none the wiser, putting their trust in doctors (who maybe should have read their medical journals a little closer).

Cholesterol is the fireman at the fire

Clearly, there is more to the cholesterol/heart disease story, because CVD occurs even with normal total cholesterol levels - it is a complicated story, involving a great deal more than just “good”and “bad”cholesterol, and yet, the simplistic, but confusing, half-told version suits those making mega-profits from lowering cholesterol with drugs, whilst disregarding the harmful consequences of those drugs.

The medical system advocates the use of drugs to lower cholesterol/blood pressure (these are the most widely used drugs According to Medco Health Solutions Inc., which manages prescription benefits for about one in five Americans) and invasive procedures (e.g. stents, angioplasties and bypass surgeries) to extend the lives of just a few of those who already have cardiovascular disease . . .

Drug solutions are not preventing CVD occurrence or solving the root problems, and the side-effects of statin drugs are increasing the risk of dying from other causes

Statins Don't Save Lives

Cholesterol is involved in heart disease, NOT as a cause

–   but rather as a consequence of chronic inflammation occurring because of ongoing damage to an artery wall

If your arteries are damaged, an inflammatory process occurs:

✔ Your blood vessels constrict to keep you from bleeding to death;

✔ Your blood becomes thicker so it can clot;

✔ Your immune system sends cells and chemicals to fight infecting viruses, bacteria and other irritants;

✔ Cells multiply to repair the damage;

✔ A scar may form, known as plaque.

✔ The plaque, thickening blood, and blood vessel constriction can increase your risk of high blood pressure and heart attacks.

If cells have been damaged, the liver is alerted to produce more cholesterol to be released into the bloodstream for delivery to the damage site (increasing LDL cholesterol levels) - LDL cholesterol (actually only the small dense ones that can squeeze through the arterial wall) becomes oxidized in the process of repairing a damaged arterial lining, developing into foam cells, which can form fatty streaks, which can later develop into artery-blocking plaque if the damage continues unheeded.

Cholesterol is the proverbial “fireman at the fire”

It makes sense then, that it would NOT be a smart move to lessen the amount of cholesterol in your blood, when your body is trying to use it to heal you –

Instead, the sensible thing to do is to stop the damage from occurring in the first place

To find out what is causing chronic heart disease (CHD) and how to deal with it, see:


CVD –A Simple Cure

Tools against CVD

High blood levels of one particular type of cholesterol Does indicate an association with atherosclerosis

–   but as a consequence of chronic inflammation occurring because of ongoing damage to an artery wall

According to a 2003 Oxford university study published in an American Heart Association journal, high Lp(a) levels are linked to a 70% probability of heart attack or stroke - Lp(a) is a very “sticky” protein made up of an LDL cholesterol with a protein (apoprotein a) wrapped around it. Data was gathered from 27 studies tracking > 5,200 people (average age 50) over 10 years, who had heart disease or survived a heart attack (the study compared the number of heart attacks suffered by individuals with the highest Lp(a) concentrations, with the number of heart attacks among those with the lowest Lp(a) readings).

Lp(a) is the “Repair Man” who arrives at the scene of injury to an artery lining to “patch up” the damage, and thus to save your life by preventing bleeding through the blood vessel wall - Lp(a) “seizes”platelets, calcium, fibrinogen (forms fibrin for blood clots) and LDL cholesterol from the blood to create a protective “repair” plaque for arteries.

Lp(a) - "The Repair Man”

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