B12 is needed to build myelin, a B12 deficiency can also cause myelin sheath damage:
Otherwise, the two most likely causes for nerve cell myelin sheath damage in M.S. involve the presence of: NEUROTOXINS and/or MICROBES
In the alternative health field, the most common theory for causing M.S. is mercury poisoning followed by a parasitic invasion of the nervous system (and particularly the brain).
Hal Huggins Website and Book "Solving the MS Mystery"
Magnesium deficiency allows heavy metal deposition in the brain. Has been shown to precede M.S., Parkinson's and Alzheimer's.
Magnesium protects cells from aluminum, mercury, lead, cadmium, beryllium and nickel. Lower than normal magnesium levels observed in CNS white matter including demyelinated plaques of MS samples.
Magnesium concentration in brains from multiple sclerosis patients. Acta Neurol. Scand Denmark 81 (3): 197-200. 1990.
If the MS patient has been infected with spirochete bacteria, then this is the likely cause (E.g. Borrelia burgdorferi transmitted by ticks causes Lyme's disease; Treponema pallidum is an agent of syphilis and yaws; Borellia recurrentis causes relapsing fever).
Oral spirochetes cause disease in man. Traditionally, dentists treated gum disease as an isolated phenomenon not damaging the whole person. We now know that gum disease is linked to common chronic illnesses. Bacteria can come from a perpetual stream of microbes from the jawbone area, as a result of periodontal disease or root canals/tooth extractions that became infected during the dental procedure. Root canals are a safe "hiding place"for microbes, until a time when they can "come out" and cause a recurrence of any disease. Levels of oral spirochetes are elevated in patients with periodontal diseases.
See: Youtube video http://www.youtube.com/watch?v=hyof5QXflos
These bacteria can go through the skin of the gums just like the syphilis spirochete bacterium (Treponema pallidum) goes through the skin of the genitals. Once inside the body they can enter the blood stream and travel to any part of the body, including blood vessel walls and the brain, and damage them. (The spirochetes found in various places in the body have been identified by PCR DNA typing to be of oral origin).
Oral spirochetes have been found in the brains of Alzheimers patients and in the plaques of heart disease patients (perhaps the link between gum disease and heart attack - the spores of spirochetes have been found in virtually all coronary artery plaques).
Spirochetes morph to a protective spore form when treated with antibiotics. When under attack, the surviving oral spirochetes will stimulate a rapid transition to a protective spore form, especially in granulation (wound-healing) tissue. A review of other spirochetal diseases traces a one hundred year trail of evidence that leads to oral spirochetes as a major contributory factor in systemic disease.
Lyme Disease and MS can follow a relapse-remitting progression, thought to result from the many different forms that spirochetes (such as Borrelia Burgdoferi) is known to take: spirochete has an elongated form (5-20um) when there is a positive environment for itsactivity, but in the presence of antibiotics it curls up defensively into an undetectable, seemingly latent granular form ( .3 - .5 ϥum)
Mattman LH, Cell Wall Deficient Forms: Stealth Pathogens (3rd Ed.), 2001, NY: CRC Press.
However, in this form it can squeeze through body pores and enter cells and organs, until when no longer threatened it can re-elongate.
Saier MH, Garcia-Lara j, The Spirochetes: Molecular and Cellular Biology. 2001, Wiltshire, UK, Horizon press
Cystic structures more common in the cerebral spinal fluid (CSF) of MS patients than in control individuals. Scientists also observed that spirochete Borrelia burgdorferi transforms to cystic forms after incubation in CSF and that they can reconvert to spirochetes if the conditions become favorable;
Philadelphia Bacteriologist Rose Ichelson reported success in cultivating the microbe, Spirochaeta myelophthora, which she has found in the spinal fluid of MS victims. She infers that MS is caused by the spirochete, and early attack on it should lead to cure or alleviation.
Ichelson, R.: Cultivation of spirochete from spinal fluids of multiple sclerosis cases and normal controls, Proc. Soc. Exper. Biol. & Med., 95:57, 1957.
Steiner observed Spirochaeta myelophthora in brain lesions from autopsied MS patients in the 1950s
Steiner G: Morphology of Spirochaeta myelophthora in M5. J Neuropathol Exp NeuroI19S4; 13: 221-229.
Older references of spirochete involvement in M.S.:
Ichelson, R.: Cultivation of spirochete from spinal fluids of multiple sclerosis cases and normal controls, Proc.
Soc. Exper. Biol. & Med., 95:57, 1957.
Schroeder, G. E.: Sclerose en plaques et spirochetes, Rev. Neurol., 41:785, 1924.
Speer, E.: Spirochatenfund im menschlichen Zentralnervensystem bei multipler Sklerose, Munch. Med. Wchschr.,
68:425, 1921.
Steiner, G.: Demonstration von Spirochaten im menschlichen Gehirn bei multipler Sklerose, Deutsche Ztschr. f. Nervenheilk., 107:112, 1928.
Steiner, G.: Acute plaques in multiple sclerosis, their pathogenetic significance and the role of spirochetes as etiological factor, J. Neuropath. & Exper. Neurol., 11:343, 1952.
Spirochetes thrive on steroids. Medication typically given to reduce neural inflammation in MS. However, the steroids could actually result in the bacteria's destruction . . . acting as bait, steroids cause spirochetes to change into a form more susceptible to T-cell attack, possibly explaining the success of steroids.
TREATMENT REQUIRES KILLING SPIROCHETE BACTERIA WITH ELECTROMEDICINE. Required because the bacteria is not in the bloodstream; electromedicine involves very low amperage electrical currents, electromagnetic waves or magnetic pulses, which can be done at home using painless, relatively affordable, electrical pulsing "tools"attached to or near the body;