GSE
Multple Sclerosis and Alternate Treatments
MS causes
First ask - Do you have a
B12
deficiency and NOT MS?
B12 is needed to build myelin,
a B12 deficiency can also cause myelin sheath
damage:
Vitamin
B12 deficiency shares common symptoms with MS.
Soreness and weakness of the legs and arms, difficulty in walking,
diminished sensory perception, difficulty in speaking, memory loss, jerking of limbs,
fatigue andparalysis;Unfortunately,
orthodox B12
tests are not accurate. The book -"Could
It Be B12? - An Epidemic of Misdiagnoses , 2nd
Edition, by Sally M. Pacholok and Jeffrey J. Stuart explains how to get a proper
diagnosis of a B12 deficiency;B12 may need to be
given by injection to be effective;
Otherwise, the two most likely causes for nerve cell
myelin sheath damage in M.S. involve the presence of: NEUROTOXINS and/or MICROBES
Neurotoxins
(especially heavy metals, but also other chemicals)
can cause MS.
Mercury poisoning
In the alternative health field, the most common theory
for causing M.S. is mercury poisoning followed by a parasitic invasion of the nervous
system (and particularly the brain).
Mercury can come from
fish, dental amalgams, vaccinations or other sources: Fish from contaminated waters.
in Norway, Japan and the state of Alaska, where they eat a lot
of ocean fish, they have high rates of MS.Dental amalgams.
The irony is that the mercury used by dentists to manufacture dental
amalgam is shipped to the dental office as a hazardous material and the amalgam
leftovers also require special precautions for disposal. Dr. Hal Huggins, a leading
world expert in biological dentistry, has treated >1000 MS patients, finding
evidence of mercury in every case.
Hal Huggins Website and Book "Solving the MS Mystery"
Magnesium
deficiency allows heavy metal deposition in the brain.
Has been shown to precede M.S., Parkinson's and Alzheimer's.
Magnesium
protects cells from aluminum, mercury, lead, cadmium, beryllium and nickel.
Lower than normal magnesium levels
observed in CNS white matter including demyelinated plaques of MS samples.
Magnesium concentration in brains from multiple sclerosis
patients. Acta Neurol. Scand Denmark 81 (3): 197-200. 1990.
(2) Microbes
(spirochete bacteria in particular) hiding inside the myelin sheath (where the immune system (I.S.) can not reach them).
The best
the I.S. can do is attack the tissue surrounding them.
This may actually cause more damage than the toxin/microbe is causing. Possible causes can be determined
through laboratory testing for pathogens, metals, and toxins.
Spirochetes
If the MS patient has been infected
with spirochete bacteria, then this is the likely cause (E.g. Borrelia
burgdorferi transmitted by ticks causes Lyme's disease; Treponema pallidum
is an agent of syphilis and yaws; Borellia recurrentis causes relapsing
fever).
Oral spirochetes cause disease in
man. Traditionally, dentists treated gum disease as an isolated
phenomenon not damaging the whole person. We now know that gum disease is linked
to common chronic illnesses. Bacteria can come from a perpetual stream of microbes
from the jawbone area, as a result of periodontal disease or root canals/tooth extractions
that became infected during the dental procedure. Root canals are a safe "hiding
place"for microbes, until a time when they can "come out" and
cause a recurrence of any disease. Levels of oral spirochetes are elevated in patients
with periodontal diseases.
See:
Youtube
video http://www.youtube.com/watch?v=hyof5QXflos
These bacteria can go through the skin of the gums just
like the syphilis spirochete bacterium (Treponema pallidum ) goes through
the skin of the genitals. Once inside the body they can enter the blood stream and
travel to any part of the body, including blood vessel walls and the brain, and
damage them. (The spirochetes found in various places in the body have been identified
by PCR DNA typing to be of oral origin).
Oral spirochetes have been found in the brains of
Alzheimers patients and in the plaques of heart disease patients (perhaps the link
between gum disease and heart attack - the spores
Invasion of Oral and Aortic Tissues by Oral Spirochete Treponema denticola in
ApoE Mice Causally Links Periodontal Disease and Atherosclerosis
Historic evidence to support a causal relationship between spirochetal
infections and Alzheimer’s disease
Spirochetes
morph to a protective spore form when treated with antibiotics.
When
under attack, the surviving oral spirochetes will stimulate a rapid transition to
a protective spore form, especially in granulation (wound-healing) tissue. A review
of other spirochetal diseases traces a one hundred year trail of evidence that leads
to oral spirochetes as a major contributory factor in systemic disease.
William D Nordquist, DDS. Oral Spirochetosis Associated with Dental Implants:
Important Clues to Systemic Disease, International Journal of Clinical Implant Dentistry,
January-April 2009;1(1):32-39
Lyme Disease and MS can follow
a relapse-remitting progression, thought to result from the many different forms
that spirochetes is known
to take
Mattman LH, Cell Wall Deficient Forms: Stealth Pathogens (3rd
Ed.), 2001, NY: CRC Press.
However, in this form it can squeeze
through body pores and enter cells and organs, until when no longer threatened it
can re-elongate.
Saier MH, Garcia-Lara j, The Spirochetes: Molecular and Cellular
Biology. 2001, Wiltshire, UK,
Horizon press
Cystic
structures more common in the cerebral spinal fluid (CSF) of MS patients than in
control individuals. Scientists also observed that spirochete
Borrelia burgdorferi transforms to cystic forms after incubation in CSF and
that they can reconvert to spirochetes if the conditions become favorable;
Brorson O,
Brorson SH, Henriksen TH, Skogen PR, Schoyen R. Association between multiple sclerosis
and cystic structures in cerebrospinal fluid. Infection 2001 Dec;29(6):315-9
Philadelphia Bacteriologist Rose Ichelson reported success in cultivating the microbe, Spirochaeta
myelophthora, which she has found in the spinal fluid of MS victims. She infers
that MS is caused by the spirochete, and early attack on it should lead to cure
or alleviation.
Ichelson, R.: Cultivation of spirochete from spinal fluids of
multiple sclerosis cases and normal controls, Proc. Soc. Exper. Biol. & Med.,
95:57, 1957.
Steiner
observed Spirochaeta myelophthora in brain lesions from autopsied MS patients in
the 1950s
Steiner G: Morphology of Spirochaeta myelophthora in M5. J
Neuropathol Exp NeuroI19S4; 13: 221-229.
Older references
of spirochete involvement in M.S.:
Henry W. Newman,
Carola Purdy, Lowell Rantz, and F.C. Hill, Jr., THE SPIROCHETE AND MULTIPLE SCLEROSIS,
Calif Med. 1958 December; 89(6): 387-389.
Ichelson, R.: Cultivation of spirochete from spinal fluids
of multiple sclerosis cases and normal controls, Proc.
Soc. Exper. Biol. & Med., 95:57, 1957.
Schroeder, G. E.: Sclerose en plaques et spirochetes, Rev.
Neurol., 41:785, 1924.
Speer, E.: Spirochatenfund im menschlichen Zentralnervensystem
bei multipler Sklerose, Munch. Med. Wchschr.,
68:425, 1921.
Steiner, G.: Demonstration von Spirochaten im menschlichen
Gehirn bei multipler Sklerose, Deutsche Ztschr. f. Nervenheilk., 107:112, 1928.
Steiner, G.: Acute plaques in multiple sclerosis, their pathogenetic
significance and the role of spirochetes as etiological factor, J. Neuropath. &
Exper. Neurol., 11:343, 1952.
Spirochetes
thrive on steroids. Medication typically given to reduce neural
inflammation in MS. However, the steroids could actually result in the bacteria's
destruction . . . acting as bait, steroids cause spirochetes to change into a form
more susceptible to T-cell attack, possibly explaining the success of steroids.
TREATMENT
REQUIRES KILLING SPIROCHETE BACTERIA WITH ELECTROMEDICINE.
Required because
the bacteria is not in the bloodstream; electromedicine involves very low amperage
electrical currents, electromagnetic waves or magnetic pulses, which can be done
at home using painless, relatively affordable, electrical pulsing "tools"attached
to or near the body;
